Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
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Frontiers Reactive oxygen species formation and its effect on CD4+ T cell-mediated inflammation
Frontiers Mitochondria, Oxidative Stress and Innate Immunity
Contribution of mito-inflammation in neuronal disorders.
The role of autophagy in pro‐inflammatory responses of microglia activation via mitochondrial reactive oxygen species in vitro - Ye - 2017 - Journal of Neurochemistry - Wiley Online Library
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IFn-λ-induced production of reactive oxygen species in mouse
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
Summary of the biology of IL-12 [14]. The main physiological producers
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Elevated type I interferon responses potentiate metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice
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