PDF) Hypothalamic loss of Snord116 recapitulates the hyperphagia
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Endocrine disorders in Prader-Willi syndrome: a model to understand and treat hypothalamic dysfunction - The Lancet Diabetes & Endocrinology
JCI - Hypothalamic loss of Snord116 recapitulates the hyperphagia of Prader-Willi syndrome
Hypothalamic AAV-BDNF gene therapy improves metabolic function and behavior in the Magel2-null mouse model of Prader-Willi syndrome: Molecular Therapy - Methods & Clinical Development
Baby food and bedtime: Evidence for opposite phenotypes from different genetic and epigenetic alterations in Prader-Willi and Angelman syndromes - Iiro Ilmari Salminen, Bernard J Crespi, Mikael Mokkonen, 2019
SnoRNA Snord116 (Pwcr1/MBII-85) Deletion Causes Growth Deficiency and Hyperphagia in Mice
PDF) Hypothalamic loss of Snord116 recapitulates the hyperphagia of Prader-Willi syndrome
PDF] Deficiency in prohormone convertase PC1 impairs prohormone processing in Prader-Willi syndrome
Baby food and bedtime: Evidence for opposite phenotypes from different genetic and epigenetic alterations in Prader-Willi and Angelman syndromes - Iiro Ilmari Salminen, Bernard J Crespi, Mikael Mokkonen, 2019
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Hypothalamic loss of Snord116 recapitulates the hyperphagia of Prader-Willi syndrome. - Abstract - Europe PMC
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